"TMT" / THE MOLECULAR THERAPY
KOCH Homeopathic Methylglyoxal/ Glyoxal vials from Germany
(Koch Catalyst Therapy)
 
"THE MOLECULAR THERAPY" Discovered by William Koch, M.D.
back in the 1920s...and promoted by Dr.Albert Szent-Gyorgi, M.D., Ph.D. (nobel prize winner for discovering Vitamin C)
 
Carbony and Di-carbonyl groups block enzymes required for cancer cell and infected cell respiration and does not harm normal cells.
 
Explaining the mechanism of action, the scientists said cancer cells required a large amount of energy providing substance called ATP (Adenosine-5-Triphosphate) for survival.
"Methylglyoxal inactivates the enzyme (Glyceraldehyde-3- Phosphate Dehydrogenase) needed for ATP production in cancer cells and thereby starves them to death. Normal cells remain unaffected."

THE KOCH TREATMENT OF CANCER
C. EVERET FIELD, M. D.
Director,  Radium Institute of New York
treat.html

http://www.williamfkoch.com/

Through extensive documentation Dr. Koch outlined the chemical processes by which disease may be reversed. Dr. Koch's research focused on the means to restore the body's oxidation mechanism back to its original vitality, thereby re-equipping the body with its innate ability to restore and maintain health, not only in cancers but also in a host of its 'allied diseases.'
This research led to Dr. Koch's development of several synthetic antitoxins: Glyoxylide, Malonide and PBQ. These catalysts became the stimulant necessary to achieve the oxidative separation of the 'host cell/pathogen integration,' when the pathogen was a virus, a carcinogen, a bacterial toxin or an incompletely burned tissue metabolite. Dr. Koch successfully defined the position of the activated amine group, the free radical, the double bond and the Carbonyl group in pathogenesis and in its correction.
 
CONTENTS:
Each Box Contains 10 Glass Vials, 2cc solution each. of 5 separate homeopathic remedies (Carbonyl & Di-Carbonyl)
Parabenzochinon, Rhodizonsaure, Carbonylgruppen SSR, Carbonylgruppen, Carbonylgruppen SSRI
 
Price: $885.00 / 3 boxes of 10 vials
INITIAL PROTOCOL REQUIRES 3-4 BOXES
Single box of 10 vials $300.00 ea.
Monitored 1 Yr.Home Protocol: $2500.00
(includes 40 vials)
 
Note: Be advised we only sell this product to our clinic patients and not the general public. If you desire to become our patient, please click this link and follow the directions:
Appointment Request & Fee Schedule
 
Because of the rarity and special care involved in hand-made manufacture of this product by the German Pharmacist, it is pricey and therefore a Special Order Item with no returns. If our supplier has current stock, it is shipped within 10 business days to us and then we ship it to the patient. If there are Customs Clearance Schedule Delays (very common these days), we will let the patient know as soon as we do. Sometimes it can be delayed by weeks. All Orders require prepayment and there are absolutely no returns or refunds.
 
RECOMMENDED USE:
Take sublingually by mouth on empty stomach or
By IM or Subcutaneous injection (preferred)
 
General 12 Month Monitored Program.
1 Vial per day for 10 days
- then -
1 Vial per week for 10 weeks
- then -
1 Vial per month for the balance or until vials are gone.
- Or -
as Directed by Healthcare Provider.
 
NOTE: Our clinic patient program includes (4) 10 vial boxes just in case one is lost or damaged since it is difficult to procure. Each vial is different so if they get out of sequence because one is broken or lost, the results won't be the same.
 
We call monthly at scheduled intervals to check on the patient's progress and to adjust the regimen if needed.
 
Vials cannot be purchased individually but only in a box of 10.

Med Hypotheses. 1997 Jun;48(6):473-6. Related Articles, Links
Does excessive adenosine 5'-triphosphate formation in cells lead to malignancy? A hypothesis on cancer.
Ray S, Ray M., Department of Biochemistry, University College of Science, University of Calcutta, India.
In biological systems, adenosine triphosphate (ATP) is the principal contributor of free energy necessary for anabolic reactions and is also a precursor of nucleic acids. Moreover, active transport of metabolites into cells is also driven by hydrolysis of ATP. So, a cell may grow, multiply and ultimately turn malignant when it has been transformed in such a manner that it produces excess ATP as compared with its usual metabolic demand. Recent studies have indicated that mitochondrial complex I and the glycolytic enzyme glyceraldehyde 3-phosphate dehydrogenase (GA3PD) may be critically altered specifically in malignant cells. So, we further propose that this excessive ATP formation may be due to altered mitochondrial complex I and GA3PD of malignant cells.
PMID: 9247887 [PubMed - indexed for MEDLINE]
Biochem J. 1997 Apr 15;323 ( Pt 2):343-8. Related Articles, Links
Selective inhibition of mitochondrial respiration and glycolysis in human leukaemic leucocytes by methylglyoxal.
Biswas S, Ray M, Misra S, Dutta DP, Ray S.
Department of Biological Chemistry, Indian Association for the Cultivation of Science, Calcutta 700 032, India.
The effect of methylglyoxal on the oxygen consumption of mitochondria of both normal and leukaemic leucocytes was tested by using different respiratory substrates and complex specific artificial electron donors and inhibitors. The results indicate that methylglyoxal strongly inhibits mitochondrial respiration in leukaemic leucocytes, whereas, at a much higher concentration, methylglyoxal fails to inhibit mitochondrial respiration in normal leucocytes. Methylglyoxal strongly inhibits ADP-stimulated alpha-oxoglutarate and malate plus NAD+-dependent respiration, whereas, at a higher concentration, methylglyoxal fails to inhibit succinate and alpha-glycerophosphate-dependent respiration. Methylglyoxal also fails to inhibit respiration which is initiated by duroquinone and cannot inhibit oxygen consumption when the N,N,N', N'-tetramethyl-p-phenylenediamine by-pass is used. NADH oxidation by sub-mitochondrial particles of leukaemic leucocytes is also inhibited by methylglyoxal. Lactaldehyde, a catabolite of methylglyoxal, can exert a protective effect on the inhibition of leukaemic leucocyte mitochondrial respiration by methylglyoxal. Methylglyoxal also inhibits l-lactic acid formation by intact leukaemic leucocytes and critically reduces the ATP level of these cells, whereas methylglyoxal has no effect on normal leucocytes. We conclude that methylglyoxal inhibits glycolysis and the electron flow through mitochondrial complex I of leukaemic leucocytes. This is strikingly similar to our previous studies on mitochondrial respiration, glycolysis and ATP levels in Ehrlich ascites carcinoma cells [Ray, Dutta, Halder and Ray (1994) Biochem. J. 303, 69-72; Halder, Ray and Ray (1993) Int. J. Cancer 54, 443-449], which strongly suggests that the inhibition of electron flow through complex I of the mitochondrial respiratory chain and inhibition of glycolysis by methylglyoxal may be common characteristics of all malignant cells.

Biochem J. 1997 Apr 15;323 ( Pt 2):343-8. Related Articles, Links
Selective inhibition of mitochondrial respiration and glycolysis in human leukaemic leucocytes by methylglyoxal.
Biswas S, Ray M, Misra S, Dutta DP, Ray S.
Department of Biological Chemistry, Indian Association for the Cultivation of Science, Calcutta 700 032, India.
http://www.biochemj.org/bj/323/0343/bj3230343.htm
Mol Cell Biochem. 1997 Jun;171(1-2):95-103. Related Articles, Links
Similar nature of inhibition of mitochondrial respiration of heart tissue and malignant cells by methylglyoxal. A vital clue to understand the biochemical basis of malignancy.
Ray S, Biswas S, Ray M.
Department of Biochemistry, University College of Science, University of Calcutta, India.http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=9201701
1: Med Hypotheses. 1997 Jun;48(6):473-6. Related Articles, Links
Does excessive adenosine 5'-triphosphate formation in cells lead to malignancy? A hypothesis on cancer.
Ray S, Ray M.
Department of Biochemistry, University College of Science, University of Calcutta, India.
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=9247887
Cell Biochem Funct. 1996 Jun;14(2):89-95. Related Articles, Links
Effects of methylglyoxal on platelet hydrogen peroxide accumulation, aggregation and release reaction.
Leoncini G, Poggi M.
Istituto Policattedra di Chimica Biologica, Universita di Genova, Italy
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=8640957
Mol Cell Biochem. 1997 Dec;177(1-2):21-6. Related Articles, Links
Inactivation of glyceraldehyde-3-phosphate dehydrogenase of human malignant cells by methylglyoxal.Ray M, Basu N, Ray S.
Department of Biological Chemistry, Indian Association for the Cultivation of Science, Jadavpur, Calcutta.http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=9450641
OTHER CITATIONS ON PUBMED...
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Display&dopt=pubmed_pubmed&from_uid=8509219


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